- Wissenschaftler
Publikationen
C5a receptor 1-/- mice are protected from the development of IgE-mediated experimental food allergy
Kordowski A, Reinicke A, Wu D, Orinska Z, Hagemann P, Huber-Lang M, Lee J, Wang Y, Hogan S, Köhl J
ALLERGY. 2019;74(4):767-779.
Ubiquitin C-terminal hydrolase L1 (UCH-L1) loss causes neurodegeneration by altering protein turnover in the first postnatal weeks
Reinicke A, Laban K, Sachs M, Kraus V, Walden M, Damme M, Sachs W, Reichelt J, Schweizer M, Janiesch P, Duncan K, Saftig P, Rinschen M, Morellini F, Meyer-Schwesinger C
P NATL ACAD SCI USA. 2019;116(16):7963-7972.
Deubiquitinating Enzyme UCH-L1 Promotes Dendritic Cell Antigen Cross-Presentation by Favoring Recycling of MHC Class I Molecules
Reinicke A, Raczkowski F, Mühlig M, Schmucker P, Lischke T, Reichelt J, Schneider E, Zielinski S, Sachs M, Jurack E, Tolosa E, Kurts C, Mittrücker H, Meyer-Schwesinger C
J IMMUNOL. 2019;203(7):1730-1742.
Ubiquitin C-Terminal Hydrolase L1 is required for regulated protein degradation through the ubiquitin proteasome system in kidney
Radón V, Czesla M, Reichelt J, Fehlert J, Hammel A, Rosendahl A, Knop J, Wiech T, Wenzel U, Sachs M, Reinicke A, Stahl R, Meyer-Schwesinger C
KIDNEY INT. 2018;93(1):110-127.
Autoantibodies against thrombospondin type 1 domain-containing 7A induce membranous nephropathy
Tomas N, Hoxha E, Reinicke A, Fester L, Helmchen U, Gerth J, Bachmann F, Budde K, Nolte F, Zahner G, Rune G, Lambeau G, Meyer-Schwesinger C, Stahl R
J CLIN INVEST. 2016;126(7):2519-32.
Alterations in the Ubiquitin Proteasome System in Persistent but Not Reversible Proteinuric Diseases
Beeken M, Lindenmeyer M, Blattner S, Radón V, Oh J, Meyer T, Hildebrand D, Schlüter H, Reinicke-Vogt A, Knop J, Vivekanandan-Giri A, Münster S, Sachs M, Wiech T, Pennathur S, Cohen C, Kretzler M, Stahl R, Meyer-Schwesinger C
J AM SOC NEPHROL. 2014;25(11):2511-25.
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